Photoreceptor cells in our eyes can adjust to both weak and strong light levels, but we still don't know exactly how they do it. Emeritus Professor Fumio Hayashi of Kobe University and his colleagues revealed that the photoreceptor protein rhodopsin forms transient clusters within the disc membranes in retina. These clusters are concentrated in the center of disc membranes, and act as platforms in the process of light to chemical signal conversion.
